| References |
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| REF 1 |
TLE1 modifies the effects of NOD2 in the pathogenesis of Crohn's disease. Gastroenterology. 2011 Sep;141(3):972-981.e1-2.
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| REF 2 |
Characterization and Genetic Analyses of New Genes Coding for NOD2 Interacting Proteins. PLoS One. 2016 Nov 3;11(11):e0165420.
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| REF 3 |
Nod2, a Nod1/Apaf-1 family member that is restricted to monocytes and activates NF-kappaB. J Biol Chem. 2001 Feb 16;276(7):4812-8.
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| REF 4 |
ITCH K63-ubiquitinates the NOD2 binding protein, RIP2, to influence inflammatory signaling pathways. Curr Biol. 2009 Aug 11;19(15):1255-63.
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| REF 5 |
The Crohn's disease protein, NOD2, requires RIP2 in order to induce ubiquitinylation of a novel site on NEMO. Curr Biol. 2004 Dec 29;14(24):2217-27.
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| REF 6 |
Inhibition of RIP2's tyrosine kinase activity limits NOD2-driven cytokine responses. Genes Dev. 2010 Dec 1;24(23):2666-77.
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| REF 7 |
The ubiquitin ligase XIAP recruits LUBAC for NOD2 signaling in inflammation and innate immunity. Mol Cell. 2012 Jun 29;46(6):746-58.
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| REF 8 |
A novel motif in the Crohn's disease susceptibility protein, NOD2, allows TRAF4 to down-regulate innate immune responses. J Biol Chem. 2011 Jan 21;286(3):1938-50.
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| REF 9 |
LUBAC-Recruited CYLD and A20 Regulate Gene Activation and Cell Death by Exerting Opposing Effects on Linear Ubiquitin in Signaling Complexes. Cell Rep. 2015 Dec 15;13(10):2258-72.
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| REF 10 |
SPATA2-Mediated Binding of CYLD to HOIP Enables CYLD Recruitment to Signaling Complexes. Cell Rep. 2016 Aug 30;16(9):2271-80.
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| REF 11 |
TAK1 is a central mediator of NOD2 signaling in epidermal cells. J Biol Chem. 2008 Jan 4;283(1):137-44.
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| REF 12 |
Reciprocal cross-talk between Nod2 and TAK1 signaling pathways. J Biol Chem. 2004 Jun 11;279(24):25876-82.
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| REF 13 |
Molecular mechanisms involved in the regulation of cytokine production by muramyl dipeptide. Biochem J. 2007 Jun 1;404(2):179-90.
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