| References |
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| REF 1 |
Prohibitin co-localizes with Rb in the nucleus and recruits N-CoR and HDAC1 for transcriptional repression. Oncogene. 2002 Dec 5;21(55):8388-96.
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| REF 2 |
Interaction of BCR-ABL with the retinoblastoma protein in Philadelphia chromosome-positive cell lines. Int J Hematol. 1997 Feb;65(2):115-21.
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| REF 3 |
Cyclin-dependent kinases regulate the antiproliferative function of Smads. Nature. 2004 Jul 8;430(6996):226-31.
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| REF 4 |
Biologic and biochemical analyses of p16(INK4a) mutations from primary tumors. J Natl Cancer Inst. 1999 Sep 15;91(18):1569-74.
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| REF 5 |
A limited screen for protein interactions reveals new roles for protein phosphatase 1 in cell cycle control and apoptosis. J Proteome Res. 2007 Mar;6(3):1165-75.
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| REF 6 |
Aromatic hydrocarbon receptor interaction with the retinoblastoma protein potentiates repression of E2F-dependent transcription and cell cycle arrest. J Biol Chem. 2000 Jan 28;275(4):2943-50.
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| REF 7 |
RBP1 recruits both histone deacetylase-dependent and -independent repression activities to retinoblastoma family proteins. Mol Cell Biol. 1999 Oct;19(10):6632-41.
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| REF 8 |
Physical interaction between pRb and cdk9/cyclinT2 complex. Oncogene. 2002 Jun 13;21(26):4158-65.
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| REF 9 |
The retinoblastoma gene product interacts with maintenance human DNA (cytosine-5) methyltransferase and modulates its activity. EMBO J. 2002 Feb 15;21(4):779-88.
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| REF 10 |
Raf-1 physically interacts with Rb and regulates its function: a link between mitogenic signaling and cell cycle regulation. Mol Cell Biol. 1998 Dec;18(12):7487-98.
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| REF 11 |
ncRNA- and Pc2 methylation-dependent gene relocation between nuclear structures mediates gene activation programs. Cell. 2011 Nov 11;147(4):773-88.
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| REF 12 |
Recruitment of the retinoblastoma protein to c-Jun enhances transcription activity mediated through the AP-1 binding site. J Biol Chem. 1999 Feb 26;274(9):5454-61.
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| REF 13 |
Acetylation control of the retinoblastoma tumour-suppressor protein. Nat Cell Biol. 2001 Jul;3(7):667-74.
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| REF 14 |
Differential specificity for binding of retinoblastoma binding protein 2 to RB, p107, and TATA-binding protein. Mol Cell Biol. 1994 Nov;14(11):7256-64.
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| REF 15 |
Exit from G1 and S phase of the cell cycle is regulated by repressor complexes containing HDAC-Rb-hSWI/SNF and Rb-hSWI/SNF. Cell. 2000 Mar 31;101(1):79-89.
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| REF 16 |
Retinoblastoma protein directly interacts with and activates the transcription factor NF-IL6. Proc Natl Acad Sci U S A. 1996 Jan 9;93(1):465-9.
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| REF 17 |
The Chk1-mediated S-phase checkpoint targets initiation factor Cdc45 via a Cdc25A/Cdk2-independent mechanism. J Biol Chem. 2006 Oct 13;281(41):30631-44.
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| REF 18 |
Structural basis for the recognition of the E2F transactivation domain by the retinoblastoma tumor suppressor. Genes Dev. 2002 Dec 15;16(24):3199-212.
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